FN Archimer Export Format
PT J
TI Low pH reduced survival of the oyster Crassostrea gigas exposed to the Ostreid herpesvirus 1 by altering the metabolic response of the host
BT 
AF Fuhrmann, Marine
   Richard, Gaëlle
   Quere, Claudie
   Petton, Bruno
   Pernet, Fabrice
AS 1:1;2:2;3:1;4:3;5:1;
FF 1:;2:;3:PDG-RBE-PFOM-LPI;4:PDG-RBE-PFOM-LPI;5:PDG-RBE-PFOM-LPI;
C1 IFREMER, LEMAR, UMR 6539, Technopole Brest Iroise, Plouzane, France
   LEMAR UMR 6539, Technopole de Brest-Iroise, Plouzané, France
   IFREMER, LEMAR, UMR 6539, Presquile Du Vivier, Argenton, France
C2 IFREMER, FRANCE
   UBO, FRANCE
   IFREMER, FRANCE
SI BREST
   ARGENTON
SE PDG-RBE-PFOM-LPI
UM LEMAR
IN WOS Ifremer UMR
   WOS Cotutelle UMR
   copubli-france
   copubli-univ-france
IF 3.224
TC 17
UR https://archimer.ifremer.fr/doc/00472/58334/60887.pdf
LA English
DT Article
DE ;Acidification;Bivalve;Disease;Environment;Metabolism;Mortality risk
AB Environmental change in the marine realm has been accompanied by emerging diseases as new pathogens evolve to take advantage of hosts weakened by environmental stress. Here we investigated how an exposure to reduced seawater pH influenced the response of the oyster Crassostrea gigas to an infection by the Ostreid herpesvirus type I (OsHV-1). Oysters were acclimated at pH 8.1 or pH 7.8 and then exposed to OsHV-1. Their survival was monitored and oyster tissues were sampled for biochemical analyses. The survival of oysters exposed to OsHV-1 at pH 7.8 was lower (33.5%) than that of their counterparts at pH 8.1 (44.8%) whereas levels of OsHV-1 DNA were similar. Energetic reserves, fatty acid composition and prostaglandin levels in oyster did not vary consistently with pH, infection or their interactions. However, there was a reduction in the activities of superoxide dismutase (SOD) and nitric oxide synthase (iNOS) in oysters at low pH, which is associated with the observed difference in survival. 
PY 2019
PD MAR
SO Aquaculture
SN 0044-8486
PU Elsevier BV
VL 503
UT 000458730400020
BP 167
EP 174
DI 10.1016/j.aquaculture.2018.12.052
ID 58334
ER

EF